This science news is interesting, but will not put food on your grocery store shelves. Everyone must have more than you think you will need for months ahead. I’ve noticed many YouTube channels have a pointer to CDC lying narrative, where they are still claiming Coronavirus is 34 times more deadly than a flu. The count was skewed and based on a small sampling of “confirmed cases.” The economic catastrophe that we are facing is way bigger than a Chinese lab. It as big as WHO which changed definition of pandemic to be any non-lethal new disease, but once they use the P word, all UN member nations must mobilize national guards. It is a set up. Having said that, for me, this Nature Magazine article (link) below gives the bio-weapon idea veracity. The website added this disclaimer which is ridiculous.
“Editors’ note, March 2020: We are aware that this story is being used as the basis for unverified theories that the novel coronavirus causing COVID-19 was engineered. There is no evidence that this is true; scientists believe that an animal is the most likely source of the coronavirus. ” Right, it came from bats. Right!
Covid -19 has been reported by multiple sources to contain HIV elements.
No one wants this to come out about Coronavirus. Spread this, it keeps getting censored.
In 2016 Nature Mag published an article about China engineering a bat coronavirus and scientists worried it would leak
In 2010 scientists in Wuhan published a study stating they successfully combined HIV with SARS. This was building on a 2007 study where they tried combining a SARS-like bat coronavirus with HIV that was not previously infectious to humans.. While they were researching the SARS-like bat Coronavirus, they were unhappy with the low rate at which it infected human cells. So they genetically altered it to make it infect human cells more effectively — by giving it an HIV (human immunodeficiency virus) outer shell, which enabled the SARS-like coronavirus to attack human cells via Ace2 receptors.
And this is significant because what I just described pretty much sums up our current Coronavirus (first study published about COVID-19, ACE2 receptors, same scientists as the study from a decade ago, Peng Zhou) .
A picture of the Lab in Wuhan, tweet from the Chinese gov. in 2018 . Where’s the hazmat suit? N95? Nope. The only BSL4 Lab in all of China, in the epicenter of the outbreak. With Asias largest virus bank
Reports that the lung damage it causes is like a combination of Sars and Aids.
Know what we have legitimate proof of? SARS accidentally leaking out of bio labs multiple times in China before.
Engineered bat virus stirs debate over risky research
Lab-made coronavirus related to SARS can infect human cells.
Editors’ note [ridiculous disclaimer], March 2020: We are aware that this story is being used as the basis for unverified theories that the novel coronavirus causing COVID-19 was engineered. There is no evidence that this is true; scientists believe that an animal is the most likely source of the coronavirus.
An experiment that created a hybrid version of a bat coronavirus — one related to the virus that causes SARS (severe acute respiratory syndrome) — has triggered renewed debate over whether engineering lab variants of viruses with possible pandemic potential is worth the risks.
In an article published in Nature Medicine1 on 9 November, scientists investigated a virus called SHC014, which is found in horseshoe bats in China. The researchers created a chimaeric virus, made up of a surface protein of SHC014 and the backbone of a SARS virus that had been adapted to grow in mice and to mimic human disease. The chimaera infected human airway cells — proving that the surface protein of SHC014 has the necessary structure to bind to a key receptor on the cells and to infect them. It also caused disease in mice, but did not kill them.
Although almost all coronaviruses isolated from bats have not been able to bind to the key human receptor, SHC014 is not the first that can do so. In 2013, researchers reported this ability for the first time in a different coronavirus isolated from the same bat population2.
The findings reinforce suspicions that bat coronaviruses capable of directly infecting humans (rather than first needing to evolve in an intermediate animal host) may be more common than previously thought, the researchers say.
But other virologists question whether the information gleaned from the experiment justifies the potential risk. Although the extent of any risk is difficult to assess, Simon Wain-Hobson, a virologist at the Pasteur Institute in Paris, points out that the researchers have created a novel virus that “grows remarkably well” in human cells. “If the virus escaped, nobody could predict the trajectory,” he says.
Creation of a chimaera
The argument is essentially a rerun of the debate over whether to allow lab research that increases the virulence, ease of spread or host range of dangerous pathogens — what is known as ‘gain-of-function’ research. In October 2014, the US government imposed a moratorium on federal funding of such research on the viruses that cause SARS, influenza and MERS (Middle East respiratory syndrome, a deadly disease caused by a virus that sporadically jumps from camels to people).
The latest study was already under way before the US moratorium began, and the US National Institutes of Health (NIH) allowed it to proceed while it was under review by the agency, says Ralph Baric, an infectious-disease researcher at the University of North Carolina at Chapel Hill, a co-author of the study. The NIH eventually concluded that the work was not so risky as to fall under the moratorium, he says.
But Wain-Hobson disapproves of the study because, he says, it provides little benefit, and reveals little about the risk that the wild SHC014 virus in bats poses to humans.
Other experiments in the study show that the virus in wild bats would need to evolve to pose any threat to humans — a change that may never happen, although it cannot be ruled out. Baric and his team reconstructed the wild virus from its genome sequence and found that it grew poorly in human cell cultures and caused no significant disease in mice.
“The only impact of this work is the creation, in a lab, of a new, non-natural risk,” agrees Richard Ebright, a molecular biologist and biodefence expert at Rutgers University in Piscataway, New Jersey. Both Ebright and Wain-Hobson are long-standing critics of gain-of-function research.
In their paper, the study authors also concede that funders may think twice about allowing such experiments in the future. “Scientific review panels may deem similar studies building chimeric viruses based on circulating strains too risky to pursue,” they write, adding that discussion is needed as to “whether these types of chimeric virus studies warrant further investigation versus the inherent risks involved”.
But Baric and others say the research did have benefits. The study findings “move this virus from a candidate emerging pathogen to a clear and present danger”, says Peter Daszak, who co-authored the 2013 paper. Daszak is president of the EcoHealth Alliance, an international network of scientists, headquartered in New York City, that samples viruses from animals and people in emerging-diseases hotspots across the globe.
Studies testing hybrid viruses in human cell culture and animal models are limited in what they can say about the threat posed by a wild virus, Daszak agrees. But he argues that they can help indicate which pathogens should be prioritized for further research attention.
Without the experiments, says Baric, the SHC014 virus would still be seen as not a threat. Previously, scientists had believed, on the basis of molecular modelling and other studies, that it should not be able to infect human cells. The latest work shows that the virus has already overcome critical barriers, such as being able to latch onto human receptors and efficiently infect human airway cells, he says. “I don’t think you can ignore that.” He plans to do further studies with the virus in non-human primates, which may yield data more relevant to humans.
- Nature Med. http://dx.doi.org/10.1038/nm.3985 (2015). et al.
- Nature 503, 535–538 (2013). et al.